Quote from trendlover:
killthesunshine think that saturated fat (CAUSE) heart disease, cancer. So he is saying no meat, no egg, no milk.
But really no cause is found, ONLY associations, correlations.
Why?
Because the animals can accumulate (dioxin) in thier (fat) in their body, and other chemical and hormone. People eat that animal and get that chemical, hormone, dioxin. This is most likely (cause) of cancers. Not the fat.
So to say to take away ALL saturated fat is wrong.
Meat has nutrients that are healthy. Yes, better to eat not too much meat, and more fruit and vegetable.
But people should find how to keep chemicals out of the animal. But what they do is say ALL animal fat is bad to eat.
your a lot like Cool Hand Luke.. you both have a whole lotta nothing
Can you read?
Stephen J. Nicholls, MBBS, PhD, FRACP, FACC*,{dagger}, Pia Lundman, MD, PhD, FESC*,{ddagger},§, Jason A. Harmer, BSc (Hons){ddagger}, Belinda Cutri, BMedSc (Hons)*, Kaye A. Griffiths, DMU{ddagger}, Kerry-Anne Rye, PhD*, Philip J. Barter, MBBS, PhD, FRACP*,|| and David S. Celermajer, MBBS, PhD, FRACP*,{ddagger},||,*
* The Heart Research Institute, Sydney, Australia
{dagger} Department of Medicine, University of Adelaide, Adelaide, Australia
{ddagger} Department of Cardiology, Royal Prince Alfred Hospital, Sydney, Australia
§ Division of Medicine, Karolinska Institutet, Danderyd University Hospital, Stockholm, Sweden
|| Department of Medicine, University of Sydney, Sydney, Australia.
Manuscript received November 23, 2005; revised manuscript received April 13, 2006, accepted April 18, 2006.
OBJECTIVES: The purpose of this study was to investigate the influence of dietary fatty acids on the anti-inflammatory properties of high-density lipoproteins (HDL) and vascular function.
BACKGROUND: The effect of dietary fatty acids on atherogenesis remains uncertain.
METHODS: Fourteen adults consumed an isocaloric meal containing either a polyunsaturated or a saturated fat on 2 occasions. The effects of post-prandial HDL on endothelial cell expression of intercellular adhesion molecule-1 (ICAM-1) and vascular cell adhesion molecule-1 (VCAM-1) were determined. Flow-mediated dilation (FMD) and microvascular reactivity were assessed before and 3 and 6 h after the meal.
RESULTS: Plasma triglycerides, insulin, and nonesterified fatty acids rose after the meals. The HDL collected 6 h after the saturated meal were less effective than HDL isolated from fasting plasma in terms of their ability to inhibit expression of ICAM-1 and VCAM-1, whereas HDL collected 6 h after the polyunsaturated meal had an inhibitory activity that was greater than that of HDL collected from fasting plasma (p < 0.004 and p = 0.01 for comparison of effect of meals on ICAM-1 and VCAM-1, respectively). Post-hyperemic microvascular flow significantly increased at 3 h after the polyunsaturated meal by 45 ± 14% and by 21 ± 11% after the saturated meal. The FMD decreased 3 h after the saturated meal by 2.2 ± 0.9% (p < 0.05 compared with baseline) and by 0.9 ± 1% after the polyunsaturated meal.
CONCLUSIONS: Consumption of a saturated fat reduces the anti-inflammatory potential of HDL and impairs arterial endothelial function. In contrast, the anti-inflammatory activity of HDL improves after consumption of polyunsaturated fat. These findings highlight novel mechanisms by which different dietary fatty acids may influence key atherogenic processes.
ONE high sat fat meal can f^ck you up. ONE!
Can you imagine TEN YEARS of 3 high fat meals per day + snacks??!! How about 20, 30, 40!!!
No wonder cardiac surgeons are millionaires 5 months out of school! They get paid by people like YOU.
Interesting... COCONUT OIL (fatty acid composition: 89.6% saturated fat, 5.8% monounsaturated, and 1.9% polyunsaturated fat) was used , last time i looked it up coconut oil isn't from animal source
