Yale Dr says HCQ has become a "political drug, not a medical drug". Could save 100k lives and alter the lockdown restrictions, dramatically. (watch Magna delete this thread
Attached below is an article on a 2005 study with Chloroqunine on SARS. Chloroquinine combined with a zinc derivative is effective in the early stages of Infection because it prevents Covid from replicating.
The real problem with the Chloroquine treatment, it is dirt cheap. The American Medical Association’s (AMA) primary purpose is to maximize healthcare provider profitability. Expensive synthetic drugs that are often derived from herbs are favored over inexpensive herbal based treatments. Often superior foreign drugs are restricted from entering the US market in an effort to maximize US pharmaceutical profits.
The US healthcare industry is about maximizing profits, not health. Long gone is the hyppocratic oath. Look at statins, for example.
Most times, when there is a controversy in a science related subject, one can either take the time to understand the science or one can follow the money. Either path leads to the conclusion that the Chloroquinine treatment is effective against Covid-SARS in the early stages of infection.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC1232869/
Chloroquine is a potent inhibitor of SARS coronavirus infection and spread
Martin J Vincent, Eric Bergeron, [...], and Stuart T Nichol
Additional article information
Abstract
Background
Severe acute respiratory syndrome (SARS) is caused by a newly discovered coronavirus (SARS-CoV). No effective prophylactic or post-exposure therapy is currently available.
Results
We report, however, that chloroquine has strong antiviral effects on SARS-CoV infection of primate cells. These inhibitory effects are observed when the cells are treated with the drug either before or after exposure to the virus, suggesting both prophylactic and therapeutic advantage. In addition to the well-known functions of chloroquine such as elevations of endosomal pH, the drug appears to interfere with terminal glycosylation of the cellular receptor, angiotensin-converting enzyme 2. This may negatively influence the virus-receptor binding and abrogate the infection, with further ramifications by the elevation of vesicular pH, resulting in the inhibition of... Click on link above to read rest of article. The article is formatted within a frame.
