oh really? LDL doesn't matter? Total cholesterol doesn't matter?
what DOES matter? does anything matter??
High LDL Cholesterol Is Not A Health 'Problem' & Other Facts
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oh really? LDL doesn't matter? Total cholesterol doesn't matter?
what DOES matter? does anything matter??
Ever notice how LDL looks a lot like LOL? I wonder if they also sound the same, and if it's a giggle or something from the "Bwahaha" family...
Crap. I wish you would of told me this earlier. I just had a bowl of Wheaties and milk laced with Sweet n Low. I bet I die in the middle of my nap I am about to take just because I had a bowl of cereal. I can't believe this. This sucks.
0
actually, he's right
cow's milk is for baby cows
goat's milk is for baby goats.
rat's milk is for err.. baby rats.
i'd fing smthg else to dilute your wheat(ies) :eek:
Marked low-density lipoprotein cholesterol reduction below current national cholesterol education program targets provides the greatest reduction in carotid atherosclerosis.
Kent SM, Coyle LC, Flaherty PJ, Markwood TT, Taylor AJ.
BACKGROUND: Current National Cholesterol Education Program (NCEP) guidelines
recognize low-density lipoprotein cholesterol (LDL-C) below 100 mg/dl as an
optimal level. Evidence supporting this is scant. Both LDL-C and C reactive
protein (CRP) are known correlates of atherosclerosis progression. HYPOTHESIS:
We examined the effect of final LDL-C and CRP obtained with statin therapy on
carotid intima-media thickness (CIMT), a valid surrogate for clinical benefit of
lipid-lowering therapies. METHODS: In a randomized, single-center trial, 161
patients were assigned to statin therapy of different potencies (pravastatin 40
mg, n = 82; atorvastatin 80 mg, n = 79). The effects on CIMT were assessed in
relationship to LDL-C and CRP levels obtained after 12 months of therapy.
RESULTS: Changes in CIMT were directly related to the final LDL-C level obtained
on statin therapy after 12 months (R = 0.219, p = 0.015). Carotid intima-media
thickness regression was seen in 61% of the subjects in the lowest quartile of
final LDL-C (< 70 mg/dl) versus 29% of the subjects with the highest quartile of
final LDL-C (> or = 114 mg/dl, p = 0.008). No threshold value was seen, with
more favorable effects on absolute change in CIMT with lower values of LDL-C
(decrease in CIMT of 0.06 +/- 0.17 mm in the lowest quartile compared with an
increase of 0.06 +/- 0.09 in the highest quartile of LDL-C, p = 0.008).
On-treatment LDL and CRP concentrations both below the group median values were
associated with the greatest likelihood of CIMT regression. CONCLUSIONS:
Regression of carotid atherosclerosis is directly related to the absolute LDL-C
level on statin therapy. The greatest regression was obtained with an LDL-C < 70
mg/dl, supporting marked LDL-C reduction to levels below current NCEP
guidelines.
Don't believe for a second that LDL doesn't matter
Kent SM, Coyle LC, Flaherty PJ, Markwood TT, Taylor AJ.
BACKGROUND: Current National Cholesterol Education Program (NCEP) guidelines
recognize low-density lipoprotein cholesterol (LDL-C) below 100 mg/dl as an
optimal level. Evidence supporting this is scant. Both LDL-C and C reactive
protein (CRP) are known correlates of atherosclerosis progression. HYPOTHESIS:
We examined the effect of final LDL-C and CRP obtained with statin therapy on
carotid intima-media thickness (CIMT), a valid surrogate for clinical benefit of
lipid-lowering therapies. METHODS: In a randomized, single-center trial, 161
patients were assigned to statin therapy of different potencies (pravastatin 40
mg, n = 82; atorvastatin 80 mg, n = 79). The effects on CIMT were assessed in
relationship to LDL-C and CRP levels obtained after 12 months of therapy.
RESULTS: Changes in CIMT were directly related to the final LDL-C level obtained
on statin therapy after 12 months (R = 0.219, p = 0.015). Carotid intima-media
thickness regression was seen in 61% of the subjects in the lowest quartile of
final LDL-C (< 70 mg/dl) versus 29% of the subjects with the highest quartile of
final LDL-C (> or = 114 mg/dl, p = 0.008). No threshold value was seen, with
more favorable effects on absolute change in CIMT with lower values of LDL-C
(decrease in CIMT of 0.06 +/- 0.17 mm in the lowest quartile compared with an
increase of 0.06 +/- 0.09 in the highest quartile of LDL-C, p = 0.008).
On-treatment LDL and CRP concentrations both below the group median values were
associated with the greatest likelihood of CIMT regression. CONCLUSIONS:
Regression of carotid atherosclerosis is directly related to the absolute LDL-C
level on statin therapy. The greatest regression was obtained with an LDL-C < 70
mg/dl, supporting marked LDL-C reduction to levels below current NCEP
guidelines.
Don't believe for a second that LDL doesn't matter
Sweden weighs in.. whoa! i'm heading for Arby's!!
Soy blocks the uptake of minerals. The soy industry is the only people that are promoting soy as healthy. The Chinese don't eat soy, they culture it and then eat it, completely different thing..
Goat's milk is a lot better than cow milk, the balance of minerals is correct for humans. Cultured goat's milk, keifer, is a super-food imo. It has the right balance of minerals and the culturing makes all the nutrients in it more bio available. Poor kids all over the world that eat keifer have great teeth..
Goat's milk is a lot better than cow milk, the balance of minerals is correct for humans. Cultured goat's milk, keifer, is a super-food imo. It has the right balance of minerals and the culturing makes all the nutrients in it more bio available. Poor kids all over the world that eat keifer have great teeth..
This is a subject very close to my heart... I am a survivor of serious heart related problems 9 years ago... I had lunch with the head of Cardiology at my reasonably prestigious hospital. The doc said "all" of us with heart problems are traceable to our genetics... (I take that "all" as a statistical "all" like 95% and not the argumentative 100% all.)
He also said that Statins and Aspirin are the two lifesavers for men of my generation. As said above, not necessarily that the statins lower cholesterol, but that the statins of 9 years ago (I am on Lipitor) change the character of the artery plaque. They make it less likely to rupture and then cause a subsequent blockage when it plugs a smaller artery downstream. He also raved about the blood thinning and anti clotting ability of aspirin. I am on a 325 mg enteric coated aspirin a day.
Don't take these drugs lightly, they are lifesavers and life extenders for some of us!
He also said that Statins and Aspirin are the two lifesavers for men of my generation. As said above, not necessarily that the statins lower cholesterol, but that the statins of 9 years ago (I am on Lipitor) change the character of the artery plaque. They make it less likely to rupture and then cause a subsequent blockage when it plugs a smaller artery downstream. He also raved about the blood thinning and anti clotting ability of aspirin. I am on a 325 mg enteric coated aspirin a day.
Don't take these drugs lightly, they are lifesavers and life extenders for some of us!