Health, Nutrition, Supplementation, What's Up With - Iodine?
https://ods.od.nih.gov/factsheets/Iodine-HealthProfessional/
The earth’s soils contain varying amounts of iodine, which in turn affects the iodine content of crops. In some regions of the world, iodine-deficient soils are common, increasing the risk of iodine deficiency among people who consume foods primarily from those areas. Salt iodization programs, which many countries have implemented, have dramatically reduced the prevalence of iodine deficiency worldwide.
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People with marginal iodine status who eat foods containing goitrogens - soy, cabbage, broccoli, cauliflower, and other cruciferous vegetables
Consumption of foods that contain goitrogens, substances that interfere with the uptake of iodine in the thyroid, can exacerbate iodine deficiency. Foods high in goitrogens include soy and cassava, cabbage, broccoli, cauliflower, and other cruciferous vegetables. Deficiencies of iron and/or vitamin A may also be goitrogenic. These issues are of concern primarily for people living in areas prone to iodine deficiency. For most people, including most of the U.S. population, who have adequate iodine intakes and eat a variety of foods, the consumption in reasonable amounts of foods containing goitrogens is not a concern.
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Iodine is a trace element that is naturally present in some foods, added to others, and available as a dietary supplement. Iodine is an essential component of the thyroid hormones thyroxine (T4) and triiodothyronine (T3). Thyroid hormones regulate many important biochemical reactions, including protein synthesis and enzymatic activity, and are critical determinants of metabolic activity. They are also required for proper skeletal and central nervous system development in fetuses and infants.
Thyroid function is primarily regulated by thyroid-stimulating hormone (TSH), also known as thyrotropin. It is secreted by the pituitary gland to control thyroid hormone production and secretion, thereby protecting the body from hypothyroidism and hyperthyroidism. TSH secretion increases thyroidal uptake of iodine and stimulates the synthesis and release of T3 and T4. In the absence of sufficient iodine, TSH levels remain elevated, leading to goiter, an enlargement of the thyroid gland that reflects the body’s attempt to trap more iodine from the circulation and produce thyroid hormones.
https://ods.od.nih.gov/factsheets/Iodine-HealthProfessional/
The earth’s soils contain varying amounts of iodine, which in turn affects the iodine content of crops. In some regions of the world, iodine-deficient soils are common, increasing the risk of iodine deficiency among people who consume foods primarily from those areas. Salt iodization programs, which many countries have implemented, have dramatically reduced the prevalence of iodine deficiency worldwide.
-
People with marginal iodine status who eat foods containing goitrogens - soy, cabbage, broccoli, cauliflower, and other cruciferous vegetables
Consumption of foods that contain goitrogens, substances that interfere with the uptake of iodine in the thyroid, can exacerbate iodine deficiency. Foods high in goitrogens include soy and cassava, cabbage, broccoli, cauliflower, and other cruciferous vegetables. Deficiencies of iron and/or vitamin A may also be goitrogenic. These issues are of concern primarily for people living in areas prone to iodine deficiency. For most people, including most of the U.S. population, who have adequate iodine intakes and eat a variety of foods, the consumption in reasonable amounts of foods containing goitrogens is not a concern.
-
Iodine is a trace element that is naturally present in some foods, added to others, and available as a dietary supplement. Iodine is an essential component of the thyroid hormones thyroxine (T4) and triiodothyronine (T3). Thyroid hormones regulate many important biochemical reactions, including protein synthesis and enzymatic activity, and are critical determinants of metabolic activity. They are also required for proper skeletal and central nervous system development in fetuses and infants.
Thyroid function is primarily regulated by thyroid-stimulating hormone (TSH), also known as thyrotropin. It is secreted by the pituitary gland to control thyroid hormone production and secretion, thereby protecting the body from hypothyroidism and hyperthyroidism. TSH secretion increases thyroidal uptake of iodine and stimulates the synthesis and release of T3 and T4. In the absence of sufficient iodine, TSH levels remain elevated, leading to goiter, an enlargement of the thyroid gland that reflects the body’s attempt to trap more iodine from the circulation and produce thyroid hormones.
