Quote from The Swordsman:
You arent listening to me, I'm not attacking you. Im saying that recent research has shown that not all saturated fats are the same. It has to do with the way in which the fatty acids are linked. Even though coconut oil is considered a saturated fat, apparently this no longer means its a bad fat to consume because its structure is not like animal fats.
anyways, i dont want to argue about it. I have heard your opinion and thats cool. Maybe others will chime in with thoughts or move onto an entirely different discussion
go drink it by the GALLON dude..
after all you got your facts off the INET, right??
to hell with peer reviewed research LOLOL
here is some more BS for you LOL:
Clin Sci (Lond). 1998 Mar;94(3):303-11.
"Dietary fish oil suppresses human colon tumour growth in athymic
mice."
Calder PC, Davis J, Yaqoob P, Pala H, Thies F, Newsholme EA.
Division of Human Nutrition, School of Biological Sciences,
University of Southampton, U.K.
"1. Human colon tumour growth, initiated by subcutaneous inoculation
of HT29 cells, was measured in athymic mice fed ad libitum on high-
fat (210 g/kg) diets rich in coconut oil (CO), olive oil (OO),
safflower oil (SO) or fish oil (FO); a low fat (LF; 25 g/kg) diet was
used as the control. In one experiment the mice were fed the
experimental diets for 3 weeks before HT29 cell inoculation and were
killed 2 weeks post-inoculation. In a second experiment the mice were
maintained on the LF diet until 4 days post-HT29 cell inoculation;
they were then fed the experimental diets for 17 days.
"2. Compared with mice fed the LF diet, tumour size was increased in
mice fed the CO, OO or SO diets for 3 weeks before HT29 cell
inoculation; FO feeding did not significantly increase tumour size.
"3. Feeding mice the CO or OO diets from 4 days post-inoculation
increased tumour growth rate and tumour size compared with feeding
the LF, SO or FO diets; tumour growth rate and size did not differ
among mice fed the latter diets.
"4. The fatty acid composition of the tumours was markedly influenced
by the fatty acid composition of the diet.
"5. We conclude that human colon tumour growth is influenced by the
type of fat consumed in the diet. Human colon tumour growth in this
model is promoted by feeding high fat diets rich in medium chain
saturated fatty acids (CO) or monounsaturated fatty acids (OO). A
high fat diet, rich in long chain n - 3 polyunsaturated fatty acids
(FO), does not promote colon tumour growth. The effect of a high fat
diet rich in n - 6 polyunsaturated fatty acids (SO) depends upon the
time at which it is fed: if fed before tumour cell inoculation such a
diet promotes tumour growth, whereas if fed once tumour growth is
initiated it does not. This suggests that n - 6 polyunsaturated fatty
acids promote the initiation of colon tumour growth, but do not exert
growth-promoting effects on colon tumours once they are established."